Osteopenia progesterone
Osteopenia progesterone background informationOsteopenia refers to bone mineral density (BMD) that is lower than normal peak BMD but not low enough to be classified as osteoporosis. Bone mineral density is a measurement of the level of minerals in the bones, which indicates how dense and strong they are. If your BMD is low compared to normal peak BMD, you are said to have osteopenia. Having osteopenia means there is a greater risk that, as time passes, you may develop BMD that is so low compared to normal its known as osteoporosis. One needs to understand the body’s process of rebuilding bone to understand the crucial role progesterone and estrogen play in bone mineral density. Bone is a living, dynamic tissue constantly being reshaped by the bone cells osteoclasts and osteoblasts. Osteoclasts and osteoblasts work together with osteoclasts reabsorbing bone while osteoblasts form bone. Osteoclasts remove bone tissue by breaking up organic bone and removing the bone mineralized matrix. This process is known as bone resorption. When osteoclasts digest this old, underlying bone, a small cavity is formed called Howship’s lacunae. The counter-part to the osteoclast is the osteoblast cell which originates in the bone marrow. Osteoblasts are responsible for bone mineralization and formation in the lacuna’s. Osteoblasts need calcium to perform their work in the process of building bone back up. Osteoblasts also regulate the amount of calcium allowed to enter the bone either by blocking calcium absorption or allowing calcium to flow across the bone and fill the lacuna’s. Osteoclasts and osteoblasts work together keeping bone in a constant state of renewal or bone remodeling, which eliminates areas of weakness, filling in cracks and fractures, thus preventing osteopenia and osteoporosis. As women age both estrogen and progesterone levels drop. Estrogen levels fall by 60-80%, while progesterone levels fall to near zero, leaving the woman in a state known as estrogen dominance. Estrogen dominance is exasperated by xenohormones, synthetic chemicals such as pesticides and plastics, which have estrogenic effects. Xenohormones combine with estrogen receptor sites to: - activate estrogenic action:
- induce extra estrogen receptors
- inhibit the ability of the liver to excrete estrogen
- inhibit the effects of progesterone
Xenohormones have become so pervasive in our environment estrogen levels are elevated to the point where they are suppressing the woman’s LH (luteinizing hormone), the pituitary hormone that signals the ovaries to release the egg and make progesterone. Our environment is so flooded with estrogen men as well as women suffer form estrogen dominance. In men there has been a 50% decrease in sperm count since 1938 and increased incidence of testicular and prostate cancer due to estrogen dominance.
Osteopenia progesterone information continued:
Osteoblasts are directly effected by the hormones estrogen and progesterone. Estrogen slows osteoblasts up, preventing them from building and remodeling bone, causing porous bone and bone loss which can lead to osteopenia. Progesterone speeds up the activity of osteoblasts, helping them to build and remodel new bone, preventing osteopenia. As women age the natural production of osteoblasts also decreases along with progesterone, increasing the probability of decreased bone mineral density, greatly increasing the probably of osteopenia. All women in permenopause or menopause should apply 28-30 mg. of real progesterone cream a day. Although natural progesterone cream is necessary it’s not sufficient to prevent osteopenia. Deficiency of certain vitamins and minerals such as calcium, magnesium, and vitamin D will also cause decreased bone mass leading to osteopenia. Not only are other vitamins and minerals necessary for healthy bones, it’s especially the form and dosage of calcium and magnesium that is crucial. Learn more at: Osteopenia progesterone at: www.ultrapms.com NOTE: all information on this page was composed and submitted by ultrapms.com, the sponsor of this page and its information.
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